Photochemotherapy, specifically the Goeckerman regimen or its clinical variants, is an intensive, specialized dermatological treatment involving the synergistic use of a topical photosensitizing or therapeutic agent (such as crude coal tar or petrolatum) and targeted ultraviolet B (UVB) phototherapy. This code, 96910, represents a single comprehensive treatment session combining these two distinct modalities. Initially developed in the 1920s by dermatologist William Goeckerman, the regimen remains a highly effective cornerstone for managing severe, recalcitrant, or generalized inflammatory skin conditions. It is predominantly utilized for generalized plaque psoriasis, but is also highly beneficial for severe atopic dermatitis (eczema), cutaneous T-cell lymphoma (such as mycosis fungoides), and prurigo nodularis that have failed to respond adequately to conventional systemic or topical therapies. The procedure begins with the meticulous application of the topical agent (coal tar or petrolatum) to the affected skin areas or the entire body surface. Coal tar possesses inherent anti-inflammatory, antipruritic, and anti-proliferative properties, which suppress the rapid turnover of epidermal cells characteristic of psoriatic plaques. Furthermore, it acts as a potent photosensitizer, significantly enhancing the skin's receptivity and responsiveness to subsequent ultraviolet irradiation. The topical agent is typically left on the skin for a specified duration, sometimes several hours or overnight in an inpatient or specialized day-treatment setting, to allow for optimal absorption and cellular interaction. Following this incubation period, the excess agent is gently removed to prevent physical blocking of the light rays, leaving only a microscopically thin, therapeutically active layer. The patient is then precisely positioned in a specialized, calibrated light booth or targeted with a focused light device to deliver a carefully calculated dose of broad-band or narrow-band Ultraviolet B (UVB) radiation. The exact dosage of UVB is determined by the patient's Fitzpatrick skin phototype, the thickness and scaling of the cutaneous plaques, and their documented previous response to phototherapy, aiming to maximize therapeutic efficacy while strictly minimizing the risk of erythema, blistering, or phototoxic burns. The synergistic combination of tar and UVB produces a robust therapeutic effect, profoundly inhibiting DNA synthesis in the hyperproliferative epidermal cells and modulating the local cutaneous immune response. This protocol is typically administered frequently, often three to five times a week, over a period of several consecutive weeks to achieve complete clearance or substantial clinical improvement of the cutaneous lesions. Rigorous clinical monitoring is absolutely essential throughout the entire treatment course to continually adjust the UVB dosimetry based on skin response and to promptly manage any localized adverse effects, ensuring both the safety and the success of the therapeutic intervention.